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Cells | Free Full-Text | Deciphering Nuclear Mechanobiology in Laminopathy | HTML
Aging: Progeria and the Lamin Connection - ScienceDirect
Hutchinson-Gilford Progeria Syndrome | IntechOpen
The Mutant Form of Lamin A that Causes Hutchinson-Gilford Progeria Is a Biomarker of Cellular Aging in Human Skin | PLOS ONE
About Progeria
Mechanics in human fibroblasts and progeria: Lamin A mutation E145K results in stiffening of nuclei - Apte - 2017 - Journal of Molecular Recognition - Wiley Online Library
Progeria, Accelerated Aging | Biochemical Mechanism of Progeria - YouTube
A progeria mutation reveals functions for lamin A in nuclear assembly, architecture, and chromosome organization | PNAS
Progeria Gene Implicated in Normal Aging | National Institutes of Health (NIH)
The association of the nuclear lamina with chromatin in normal and... | Download Scientific Diagram
Accumulation of mutant lamin A causes progressive changes in nuclear architecture in Hutchinson–Gilford progeria syndrome | PNAS
Phosphorylated Lamin A/C in the Nuclear Interior Binds Active Enhancers Associated with Abnormal Transcription in Progeria - ScienceDirect
Figure 1 | Pharmacotherapy to gene editing: potential therapeutic approaches for Hutchinson–Gilford progeria syndrome | SpringerLink
Progeria
Research – Gonzalo Lab
Vía de síntesis de la lamina-A (izquierda). A la derecha se muestra la... | Download Scientific Diagram
Progeria: A Paradigm for Translational Medicine: Cell
Projects – Buchwalter Lab
A lamin A protein isoform overexpressed in Hutchinson–Gilford progeria syndrome interferes with mitosis in progeria and normal cells | PNAS
Normal and Hutchinson-Gilford progeria syndrome nuclei differ in... | Download Scientific Diagram
The Defective Nuclear Lamina in Hutchinson-Gilford Progeria Syndrome Disrupts the Nucleocytoplasmic Ran Gradient and Inhibits Nuclear Localization of Ubc9 | Molecular and Cellular Biology
Molecular insights into the premature aging disease progeria | SpringerLink
Phosphorylated Lamin A/C in the Nuclear Interior Binds Active Enhancers Associated with Abnormal Transcription in Progeria - ScienceDirect